[Completed]
For the first time in the world, we have demonstrated through molecular simulations the ability of specific compounds to:
Alter the structure of proteins responsible for mitochondrial fragmentation, preventing them from binding to mitochondria.
Activate receptors that restore energy flow to the endoplasmic reticulum.
Block factors that suppress mitochondrial biogenesis, forming a collective defense against them.
This study utilized a custom-developed computational pipeline based on Stanford University's MD engine, optimizing the same screening workflow used by pharmaceutical companies at an unprecedented level of efficiency.
We quantitatively evaluated the physiological effects of four key compounds through computer simulations.
 
Physiological Mechanisms Evaluated
① Activation of mitochondrial biogenesis and restoration of energy flow
② Inhibition of mitochondrial fragmentation, a major obstacle in long COVID recovery
③ Structural deformation of SARS-CoV-2 viral proteins (and their complexes) that abnormally activate these fragmentation pathways
 
Intended Audience
🟢 Science enthusiast  
🟢 Individuals in good health who wish to enhance cognitive performance and energy levels
🟢 Those experiencing cognitive or physical decline following COVID-19 infection
 
Who Should Exercise Caution? (⚠ Requires additional guidance)
🔴 Individuals sensitive to hormonal balance
🔴 Those taking medications affecting blood sugar or blood clotting
🔴 Individuals with high uric acid levels or a history of gout
🔴 Those with compromised liver function
 
Disclaimer
This report is intended for educational and informational purposes only and does not constitute medical advice, diagnosis, or treatment.
Before making any decisions regarding supplements or health interventions, please consult with a physician or healthcare professional.